Identifying Table Structure in Documents using Conditional Generative Adversarial Networks

In many industries, as well as in academic research, information is primarily transmitted in the form of unstructured documents (this article, for example). Hierarchically-related data is rendered as tables, and extracting information from tables in such documents presents a significant challenge. Many existing methods take a bottom-up approach, first integrating lines into cells, then cells into rows or columns, and finally inferring a structure from the resulting 2-D layout. But such approaches neglect the available prior information relating to table structure, namely that the table is merely an arbitrary representation of a latent logical structure. We propose a top-down approach, first using a conditional generative adversarial network to map a table image into a standardised `skeleton' table form denoting approximate row and column borders without table content, then deriving latent table structure using xy-cut projection and Genetic Algorithm optimisation. The approach is easily adaptable to different table configurations and requires small data set sizes for training.

Extracting Tables from Documents using Conditional Generative Adversarial Networks and Genetic Algorithms

Extracting information from tables in documents presents a significant challenge in many industries and in academic research. Existing methods which take a bottom-up approach of integrating lines into cells and rows or columns neglect the available prior information relating to table structure. Our proposed method takes a top-down approach, first using a generative adversarial network to map a table image into a standardised `skeleton' table form denoting the approximate row and column borders without table content, then fitting renderings of candidate latent table structures to the skeleton structure using a distance measure optimised by a genetic algorithm.

Synaptic Scaling Balances Learning in a Spiking Model of Neocortex

Learning in the brain requires complementary mechanisms: potentiation and activity-dependent homeostatic scaling. We introduce synaptic scaling to a biologically-realistic spiking model of neocortex which can learn changes in oscillatory rhythms using STDP, and show that scaling is necessary to balance both positive and negative changes in input from potentiation and atrophy. We discuss some of the issues that arise when considering synaptic scaling in such a model, and show that scaling regulates activity whilst allowing learning to remain unaltered.

Effects of Compensation, Connectivity and Tau in a Computational Model of Alzheimer's Disease

This work updates an existing, simplistic computational model of Alzheimer's Disease (AD) to investigate the behaviour of synaptic compensatory mechanisms in neural networks with small-world connectivity, and varying methods of calculating compensation. It additionally introduces a method for simulating tau neurofibrillary pathology, resulting in a more dramatic damage profile. Small-world connectivity is shown to have contrasting effects on capacity, retrieval time, and robustness to damage, whilst the use of more easily-obtained remote memories rather than recent memories for synaptic compensation is found to lead to rapid network damage.